She's a COVID victim
Politicians, doctors, pharmacists, scientists, researchers, MsM, there seem to be more liars than not.
So we fall back to total deaths. Screen cap:
Richplanet Show 282 The Corona Scam
Captap NaturalWisdom www.rumormill.news/145048
There are other agendas in play more important than Gates and his poisononous vaccines.
Share. Firefox, right click, download. This copy is 142.5MB, the originals is 240.5.
Bitchute March 4th, 2020
What you need to know about Coronavirus, "Pandemics", 5G, 60 ghz millimetre waves and vaccines
Part 2 out now: https://www.bitchute.com/video/7NuuW1b9Z9rR/
Part 3 also out now: https://www.bitchute.com/video/4iFI8whxumKJ/
Part 4 https://www.bitchute.com/video/9z0E4ZxaQbbr/
Dana Ashlie: Corona Virus: https://www.youtube.com/watch?v=CtfqUtW_8AA
the Kissinger report 1974: https://pdf.usaid.gov/pdf_docs/PCAAB500.pdf
Pasteur, Plagiarist, Imposter pdf: http://www.mnwelldir.org/docs/history/biographies/Bechamp-or-Pasteur.pdf
Fasting and Man’s Correct Diet R.B. Pearson: https://books.google.dk/books?id=u7-h-oz785sC&printsec=frontcover&hl=da&source=gbs_ge_summary_r&cad=0#v=onepage&q&f=false
Annie Logical, Pandemic Bonds and World Bank: https://www.vigiliae.org/pandemic-bonds-and-world-bank/
Dr Stefan Lanka, Virologist, Microbiologist, HIV Hoax: https://www.youtube.com/watch?v=-btqQa-wXno
You cannot Catch Bugs, Pasteur Debunked: https://www.healingnaturallybybee.com/you-cannot-catch-bugs-germs-bacteria-or-candidafungi/
Aajonus Vonderplanitz full Transcript, Superhuman Radio: http://whale.to/a/aajonus-interview-apr-2009.pdf
Posted April 18th 2020
Written By: Sayer Ji, Founder
copyrighted by GreenMedInfo LLC, 2020
Sayer Ji, author of REGENERATE, discusses the controversy around the similarity between viruses and extracellular vesicles called exosomes, as well as the possibility that what appears to be contagion-based viral infectivity may also be understood as a xenohormetic response to toxicant or EMF-associated cell damageCited or relevant articles, videos, and research papers include:
- Let's Get Real About This So-Called "Plan-Demic"
- Why Everything You Learned About Viruses is WRONG
- Plant-Derived Exosomes as Cross-Species Messengers and Beacons of Epigenetics
- VIDEO: Coronavirus Fear, Germ Theory, Exosomes, and Resiliency
- VIDEO: Andrew Kaufman on Exosomes, Coronavirus and Germ Theory
- Amazing Food Science Discovery: Edible Plants 'Talk' To Animal Cells, Promote Healing
- Exosome studies on Greenmedinfo (110 abstracts)
- How the Microbiome Undermines the Ego, Vaccine Policy and Patriarchy
- The GMO Agenda Takes a Menacing Leap Forward with RNAi Corn
- No Sex Required: Body Cells Transfer Genetic Info Directly Into Sperm Cells, Amazing Study Finds
- The Dark and Light Side of Food As Information Dietary RNAs
- COVID-19 Related Articles
- Exogenous exosomes from mice with acetaminophen-induced liver injury promote toxicity in the recipient hepatocytes and mice. Sci Rep. 2018 Oct 30;8(1):16070. doi: 10.1038/s41598-018-34309-7. Cho YE
- HIV As Trojan Exosome: Immunological Paradox Explained? Front Immunol. 2017; 8: 1715.Published online 2017 Dec 1. James E. K. Hildret ***
Captap Hobie www.rumormill.news/145107
***14. HIV As Trojan Exosome: Immunological Paradox Explained?
James E. K. Hildreth
The HIV pandemic is still a major global challenge, despite the widespread availability of antiretroviral drugs. An effective vaccine would be the ideal approach to bringing the pandemic to an end. However, developing an effective HIV vaccine has proven to be an elusive goal. Three major human HIV vaccine trials revealed a strong trend toward greater risk of infection among vaccine recipients versus controls. A similar observation was made in a macaque SIV vaccine study. The mechanism explaining this phenomenon is not known. Here, a model is presented that may explain the troubling results of vaccine studies and an immunological paradox of HIV pathogenesis: preferential infection of HIV-specific T cells. The central hypothesis of this perspective is that as “Trojan exosomes” HIV particles can directly activate HIV-specific T cells enhancing their susceptibility to infection. Understanding the biology of HIV as an exosome may provide insights that enable novel approaches to vaccine development.
Carnathan et al. (1) employed the SIV/macaque model to evaluate immunization regimens under investigation in preclinical and clinical HIV vaccine studies. Their vaccine vectors did not include the envelope gene and could not induce neutralizing antibodies. Thus, the study specifically evaluated the role of cellular immune responses in protection against virus acquisition or control of replication after infection. All groups of animals showed SIV-specific CD8 T cells, but there was no correlation between the function or number of such cells and infection after rectal virus challenge. Notably, there were much higher levels of activated memory CD4 T cells in rectal biopsies from infected animals than from animals that remained negative after challenge. These data support the hypothesis that mucosal levels of activated CD4+ CCR+ T cells in virus-exposed animals predict the risk of virus acquisition.
It is well established that HIV preferentially infects activated memory CD4 T cells (2). For this reason, any factor that increases the number of such cells in mucosal tissues exposed to HIV may increase the risk of virus acquisition. Other sexually transmitted infections that cause accumulation of mucosal inflammatory cells are among such factors (3). Preferential infection of activated CD4 T cells may also explain sequential loss of CD4 T cells of defined specificities. For example, Loré et al. demonstrated that HIV-infected dendritic cells (DCs) can present CMV antigens and activate CMV-specific CD4 T cells. In their study, HIV was transmitted to activated CMV-specific T cells but not to non-responding T cells (4). These observations raise the possibility that any HIV vaccine that induces strong CD4 T cell responses may increase risk of transmission. As noted above, results from three major clinical vaccine trials evaluating multiple vaccine regimens—the HVTN-505, Phambili, and STEP trials—showed a strong trend toward greater risk of HIV acquisition among vaccine recipients versus placebo recipients (5–7). The mechanism explaining this troubling observation is unknown, but may be simply that the vaccines increased the pool of memory CD4 T cells in mucosal tissues. Interestingly, when Douek et al. examined the specificity of HIV-infected memory T cells from infected individuals, the data revealed that HIV preferentially infects HIV-specific CD4 T cells (8). These results suggest a peculiar immunological paradox for HIV: targeted infection of the very T cells that are programmed to respond to it. This phenomenon also predicts that vaccination regimens that increase the pool of mucosal HIV-specific CD4 T cells may result in greater risk of virus acquisition consistent with the trend toward higher risk of infection among HIV vaccine recipients (5–7).
Conclusion, vaccines are a really bad idea.